Research
Epitranscriptomics & Cancer Adaptation : A.David

Activities

Our research work focuses on the contribution of post-transcriptional mechanisms on cancer cell adaptation, in particular RNA epigenetic & translational control.

More..

Zotero public









Cancel


15 Items
Title  Creator  Group name  Publishing year 
Fibroblast-derived prolargin is a tumor suppressor in hepatocellular carcinoma Chiavarina et al. EquipeJC 2022-03
Metastatic colorectal cancer cells maintain the TGF? program and use TGFBI to fuel angiogenesis Chiavarina et al. EquipeVC 2021
Transforming growth factor beta-induced, an extracellular matrix interacting protein, enhances glycolysis and promotes pancreatic cancer cell migration Costanza et al. EquipeAT Mar 04, 2019
Methylglyoxal, a glycolysis metabolite, triggers metastasis through MEK/ERK/SMAD1 pathway activation in breast cancer Nokin et al. EquipeAT 01 23, 2019
Murine stroma adopts a human-like metabolic phenotype in the PDX model of colorectal cancer and liver metastases Blomme et al. EquipeAT Mar 2018
Innovative methodology for the identification of soluble biomarkers in fresh tissues Costanza et al. EquipeAT Feb 13, 2018
Methylglyoxal-Mediated Stress Correlates with High Metabolic Activity and Promotes Tumor Growth in Colorectal Cancer Chiavarina et al. EquipeAT Jan 21, 2017
[Asporin: the protective wall against triple-negative breast cancer] Blomme et al. EquipeAT Nov 2016
New role of osteopontin in DNA repair and impact on human glioblastoma radiosensitivity Henry et al. EquipeAT Sep 27, 2016
Myoferlin plays a key role in VEGFA secretion and impacts tumor-associated angiogenesis in human pancreas cancer Fahmy et al. EquipeAT Feb 01, 2016
Myoferlin is a novel exosomal protein and functional regulator of cancer-derived exosomes Blomme et al. EquipeAT Dec 13, 2016
Methylglyoxal, a glycolysis side-product, induces Hsp90 glycation and YAP-mediated tumor growth and metastasis Nokin et al. EquipeAT 10 19, 2016
Targeting osteopontin suppresses glioblastoma stem-like cell character and tumorigenicity in vivo Lamour et al. EquipeAT Sep 01, 2015
Histone deacetylases and cancer-associated angiogenesis: current understanding of the biology and clinical perspectives Turtoi et al. EquipeAT 2015
Triple negative tumors accumulate significantly less methylglyoxal specific adducts than other human breast cancer subtypes Chiavarina et al. EquipeAT Jul 30, 2014
15 Items


Who are we ?

Located in the Parc Euromédecine, the Institut de Recherche en Cancérologie de Montpellier (U1194) is located on the Val d'Aurelle Campus (ICM, Institut régional du Cancer Montpellier/ Val d'Aurelle).

U1194 Research Centre supported by Inserm, the University of Montpellier and the Institut du Cancer de Montpellier (ICM)

More                                                                                                                 Join us

Contact us

Institut de Recherche en Cancérologie de
Montpellier (U1194) - Campus Val d'Aurelle

208 avenue des Apothicaires
34298 Montpellier cedex 5 FRANCE

 

insermUM1ICM

© Institut de Recherche en Cancérologie de Montpellier - 2011 - Tous droits réservés - Mentions légales - Connexion - Conception : ID Alizés